American Journal of Physiology-Cell Physiology

Journal Information
ISSN / EISSN : 03636143 / 15221563
Current Publisher: American Physiological Society (10.1152)
Total articles ≅ 11,650
Google Scholar h5-index: 50
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Latest articles in this journal

Sophie I. Hamstra, Kennedy C. Whitley, Ryan W. Baranowski, Nigel Kurgan, Jessica L. Braun, Holt N. Messner, Val A. Fajardo
American Journal of Physiology-Cell Physiology; doi:10.1152/ajpcell.00318.2020

The publisher has not yet granted permission to display this abstract.
Songtao Shi, Hongli Li
American Journal of Physiology-Cell Physiology; doi:10.1152/ajpcell.00479.2019

The publisher has not yet granted permission to display this abstract.
An-Ci Siao, Yen-Yue Lin, Li-Jane Shih, Yi-Wei Tsuei, Chih-Pin Chuu, Yow-Chii Kuo, Yung-Hsi Kao
American Journal of Physiology-Cell Physiology; doi:10.1152/ajpcell.00491.2019

The publisher has not yet granted permission to display this abstract.
Bei Chen, Hongen Xu, Yanfang Mi, Wei Jiang, Dan Guo, Jinhui Zhang, Yulin Zhao, Wenxue Tang
American Journal of Physiology-Cell Physiology; doi:10.1152/ajpcell.00483.2019

The publisher has not yet granted permission to display this abstract.
Emma Teal, Martha Dua-Awereh, Sabrina T. Hirshorn, Yana Zavros
American Journal of Physiology-Cell Physiology; doi:10.1152/ajpcell.00415.2019

The publisher has not yet granted permission to display this abstract.
Guangdong Yang
American Journal of Physiology-Cell Physiology, Volume 319; doi:10.1152/ajpcell.00187.2020

Abstract:
The outbreak of COVID-19 pneumonia caused by a new coronavirus (severe acute respiratory syndrome coronavirus 2, SARS-CoV-2) is posing a global health emergency and has led to more than 380,000 deaths worldwide. The cell entry of SARS-CoV-2 depends on two host proteins angiotensin-converting enzyme 2 (ACE2) and transmembrane protease serine 2 (TMPRSS2). There is currently no vaccine available and also no effective drug for the treatment of COVID-19. Hydrogen sulfide (H2S) as a novel gasotransmitter has been shown to protect against lung damage via its anti-inflammation, antioxidative stress, antiviral, prosurvival, and antiaging effects. In light of the research advances on H2S signaling in biology and medicine, this review proposed H2S as a potential defense against COVID-19. It is suggested that H2S may block SARS-CoV-2 entry into host cells by interfering with ACE2 and TMPRSS2, inhibit SARS-CoV-2 replication by attenuating virus assembly/release, and protect SARS-CoV-2-induced lung damage by suppressing immune response and inflammation development. Preclinical studies and clinical trials with slow-releasing H2S donor(s) or the activators of endogenous H2S-generating enzymes should be considered as a preventative treatment or therapy for COVID-19.
Huocong Huang, Rolf A Brekken
American Journal of Physiology-Cell Physiology, Volume 319; doi:10.1152/ajpcell.00079.2020

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Lennart Kuck, Jason N. Peart, Michael J. Simmonds
American Journal of Physiology-Cell Physiology, Volume 319; doi:10.1152/ajpcell.00210.2020

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Danielle Hiam, Severine Lamon
American Journal of Physiology-Cell Physiology, Volume 319; doi:10.1152/ajpcell.00175.2020

Evelyn Zacharewicz, Ming Kalanon, Robyn M. Murphy, Aaron P. Russell, Severine Lamon
American Journal of Physiology-Cell Physiology, Volume 319; doi:10.1152/ajpcell.00172.2020

The publisher has not yet granted permission to display this abstract.
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