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(searched for: doi:10.4008/*)
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Sciprofile linkSelim Kurtoğlu, Esad Köklü, Nihal Hatipoğlu, Mehmet Emre Atabek
Journal of Clinical Research in Pediatric Endocrinology, Volume 1, pp 1-7; doi:10.4008/jcrpe.v1i1.14

Abstract: Background: Zinc (Zn) plays a central role in the activation of numerous enzyme systems that synthesize and degrade bioactive peptides. Some of these bioactive peptides, also called neuropeptides, are involved in the regulation of food intake.
Sciprofile linkOzgur Pirgon, Mehmet Emre Atabek, Bülent Oran, Rıdvan Güçlü
Journal of Clinical Research in Pediatric Endocrinology, Volume 1, pp 62-71; doi:10.4008/jcrpe.v1i2.9

Abstract: Objective: Women with Turner syndrome (TS) have greater carotid artery intima-media thickness (cIMT) known to be a risk factor for atherosclerosis in adults. To determine whether there are risk factors for atherosclerosis in children with TS, we compared cIMT, anthropometric and metabolic parameters between children with TS and healthy controls.
Sciprofile linkSelim Kurtoğlu, Dilek Coban, Mustafa Ali Akın, Sciprofile linkLeyla Akın, Ali Yıkılmaz
Journal of Clinical Research in Pediatric Endocrinology, Volume 1; doi:10.4008/jcrpe.v1i4.54

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Sciprofile linkHayrullah Alp, Mehmet Emre Atabek, Sciprofile linkOzgur Pirgon
Journal of Clinical Research in Pediatric Endocrinology, Volume 1; doi:10.4008/jcrpe.v1i4.45

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Journal of Clinical Research in Pediatric Endocrinology, Volume 1; doi:10.4008/jcrpe.v1i4.50

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Journal of Clinical Research in Pediatric Endocrinology, Volume 1; doi:10.4008/jcrpe.v1i4.57

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Journal of Clinical Research in Pediatric Endocrinology, Volume 1; doi:10.4008/jcrpe.v1i4.3

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Fima Lifshitz
Journal of Clinical Research in Pediatric Endocrinology, Volume 1; doi:10.4008/jcrpe.v1i4.39

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Sciprofile linkSema Kabataş Eryılmaz, Firdevs Baş, Ali Satan, Feyza Darendeliler, Rüveyde Bundak, Hülya Günöz, Nurçin Saka
Journal of Clinical Research in Pediatric Endocrinology, Volume 1, pp 151-154; doi:10.4008/jcrpe.v1i3.46

Abstract: 22q11 deletion is one of the most frequently encountered genetic syndromes. The phenotypic spectrum shows a wide variability. We report a boy who presented at age 11.9 years with seizures due to hypocalcemia as a result of hypoparathyroidism. FISH analysis revealed a heterozygote deletion at 22q11.2. Positive findings for the syndrome were delayed speech development due to velofacial dysfunction, recurrent croup attacks in early childhood due to latent hypocalcemia and mild dysmorphic features. The findings of this patient indicate that 22q11 deletion syndrome may present with a wide spectrum of clinical findings and that this diagnosis needs to be considered even in patients of older ages presenting with hypocalcemia.