miR-204-5p Represses Bone Metastasis via Inactivating NF-κB Signaling in Prostate Cancer
Open Access
- 18 September 2019
- journal article
- research article
- Published by Elsevier BV in Molecular Therapy Nucleic Acids
- Vol. 18, 567-579
- https://doi.org/10.1016/j.omtn.2019.09.008
Abstract
No abstract availableFunding Information
- National Natural Science Foundation of China (81872172, 81660362, 81503281, 81702671, 81502219)
- Science and Technology Planning Project of Health and Family Planning Commission of Jiangxi Province (20181026)
- Talent Grant of Sun Yat-Sen University (17ykpy30)
This publication has 73 references indexed in Scilit:
- VHL-Regulated MiR-204 Suppresses Tumor Growth through Inhibition of LC3B-Mediated Autophagy in Renal Clear Cell CarcinomaCancer Cell, 2012
- Expanding role of ubiquitination in NF-κB signalingCell Research, 2010
- Deubiquitinases in the regulation of NF-κB signalingCell Research, 2010
- Androgen Receptor Requires JunD as a Coactivator to Switch on an Oxidative Stress Generation Pathway in Prostate Cancer CellsCancer Research, 2010
- Signaling to NF- B: Regulation by UbiquitinationCold Spring Harbor Perspectives in Biology, 2009
- Shared Principles in NF-κB SignalingCell, 2008
- Nuclear factor-κB in cancer development and progressionNature, 2006
- NF-κB: linking inflammation and immunity to cancer development and progressionNature Reviews Immunology, 2005
- De-ubiquitination and ubiquitin ligase domains of A20 downregulate NF-κB signallingNature, 2004
- Activation of the IκB Kinase Complex by TRAF6 Requires a Dimeric Ubiquitin-Conjugating Enzyme Complex and a Unique Polyubiquitin ChainCell, 2000