Axl as a downstream effector of TGF-β1 via PI3K/Akt-PAK1 signaling pathway promotes tumor invasion and chemoresistance in breast carcinoma
- 21 October 2014
- journal article
- retracted article
- Published by Springer Science and Business Media LLC in Tumor Biology
- Vol. 36 (2), 1115-1127
- https://doi.org/10.1007/s13277-014-2677-3
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Glycoxidised LDL Induced the Upregulation of Axl Receptor Tyrosine Kinase and Its Ligand in Mouse Mesangial CellsPLOS ONE, 2012
- Mer or Axl receptor tyrosine kinase inhibition promotes apoptosis, blocks growth and enhances chemosensitivity of human non-small cell lung cancerOncogene, 2012
- Axl glycosylation mediates tumor cell proliferation, invasion and lymphatic metastasis in murine hepatocellular carcinomaWorld Journal of Gastroenterology, 2012
- HAb18G/CD147 promotes epithelial–mesenchymal transition through TGF-β signaling and is transcriptionally regulated by SlugOncogene, 2011
- The Axl receptor tyrosine kinase is an adverse prognostic factor and a therapeutic target in esophageal adenocarcinomaCancer Biology & Therapy, 2010
- TGF-β1–induced expression of human Mdm2 correlates with late-stage metastatic breast cancerJCI Insight, 2010
- Fonctions nouvelles de Gas-6 et de la protéine Smédecine/sciences, 2007
- The evolution of phosphatidylinositol 3-kinases as regulators of growth and metabolismNature Reviews Genetics, 2006
- Clinical significance of AXL kinase family in gastric cancer.2002
- Estrogen dependent expression of the receptor tyrosine kinase axl in normal and malignant human breastAnnals of Oncology, 2001