The C57Bl/6J mouse strain is more susceptible to angiotensin II-induced aortic aneurysm formation than C57Bl/6N
- 1 December 2020
- journal article
- research article
- Published by Elsevier BV in Atherosclerosis
- Vol. 318, 8-13
- https://doi.org/10.1016/j.atherosclerosis.2020.11.032
Abstract
No abstract availableKeywords
Funding Information
- Heidelberger Stiftung Chirurgie
- Deutsche Forschungsgemeinschaft
This publication has 6 references indexed in Scilit:
- Critical Role of Cytosolic DNA and Its Sensing Adaptor STING in Aortic Degeneration, Dissection, and RuptureJournal of the American College of Cardiology, 2020
- Updates of Recent Aortic Aneurysm ResearchArteriosclerosis, Thrombosis, and Vascular Biology, 2019
- Absence of Nicotinamide Nucleotide Transhydrogenase in C57BL/6J Mice Exacerbates Experimental AtherosclerosisJournal of Vascular Research, 2018
- Animal Models Used to Explore Abdominal Aortic Aneurysms: A Systematic ReviewEuropean Journal of Vascular and Endovascular Surgery, 2016
- Redox signaling in cardiovascular pathophysiology: A focus on hydrogen peroxide and vascular smooth muscle cellsRedox Biology, 2016
- A comparative phenotypic and genomic analysis of C57BL/6J and C57BL/6N mouse strainsGenome Biology, 2013