Game of clones: How measles remodels the B cell landscape
- 1 November 2019
- journal article
- editorial
- Published by American Association for the Advancement of Science (AAAS) in Science Immunology
- Vol. 4 (41)
- https://doi.org/10.1126/sciimmunol.aaz4195
Abstract
B cell receptor sequencing sheds light on how measles cripples the immune system long after recovery from clinical disease (see related Research Articles by Petrova et al. and Mina et al.).Keywords
This publication has 10 references indexed in Scilit:
- Incomplete genetic reconstitution of B cell pools contributes to prolonged immunosuppression after measlesScience Immunology, 2019
- Measles virus infection diminishes preexisting antibodies that offer protection from other pathogensScience, 2019
- Affinity war: forging immunoglobulin repertoiresCurrent Opinion in Immunology, 2019
- Breaching peripheral tolerance promotes the production of HIV-1–neutralizing antibodiesThe Journal of Experimental Medicine, 2017
- Transitional B Cells in Early Human B Cell Development – Time to Revisit the Paradigm?Frontiers in Immunology, 2016
- Complex Antigens Drive Permissive Clonal Selection in Germinal CentersImmunity, 2016
- Long-term measles-induced immunomodulation increases overall childhood infectious disease mortalityScience, 2015
- Measles Immune Suppression: Lessons from the Macaque ModelPLoS Pathogens, 2012
- Predominant Autoantibody Production by Early Human B Cell PrecursorsScience, 2003
- Maintenance of Serological Memory by Polyclonal Activation of Human Memory B CellsScience, 2002