Neutrophils, NETs, NETosis and their paradoxical roles in COVID-19
Open Access
- 11 May 2020
- journal article
- Published by Heighten Science Publications Corporation in Journal of Stem Cell Therapy and Transplantation
- Vol. 4 (1), 003-010
- https://doi.org/10.29328/journal.jsctt.1001020
Abstract
Systemic sclerosis (SScl) is an autoimmune disorder of unknown aetiology, characterised by fibrosis and microvascular injury of the affected organs. The hallmark of the disease is thickening and tightness of the skin and the subcutaneous tissue. SScl can affect virtually any organ systems, most importantly the skin, blood vessels, lungs, kidneys, gastrointestinal tract, and the heart [1].Keywords
This publication has 84 references indexed in Scilit:
- Neutrophil heterogeneity in health and disease: a revitalized avenue in inflammation and immunityOpen Biology, 2012
- Neutrophil extracellular traps: Is immunity the second function of chromatin?The Journal of cell biology, 2012
- Hypochlorous acid regulates neutrophil extracellular trap release in humansClinical and Experimental Immunology, 2011
- Excessive Neutrophils and Neutrophil Extracellular Traps Contribute to Acute Lung Injury of Influenza PneumonitisThe American Journal of Pathology, 2011
- PAD4-Mediated Neutrophil Extracellular Trap Formation Is Not Required for Immunity against Influenza InfectionPLOS ONE, 2011
- Myeloperoxidase is required for neutrophil extracellular trap formation: implications for innate immunityBlood, 2011
- Statins Enhance Formation of Phagocyte Extracellular TrapsCell Host & Microbe, 2010
- Neutrophil extracellular trap cell death requires both autophagy and superoxide generationCell Research, 2010
- Novel cell death program leads to neutrophil extracellular trapsThe Journal of cell biology, 2007
- Neutrophil Extracellular Traps Kill BacteriaScience, 2004